Last reviewed · How we verify

metoprolol/HCT

Abbott · Phase 3 active Small molecule

metoprolol/HCT is a Beta-blocker Small molecule drug developed by Abbott. It is currently in Phase 3 development for Hypertension, Atrial fibrillation for stroke prevention.

Metoprolol/HCT is a beta-blocker that works by blocking the effects of the hormone epinephrine, and also has a diuretic effect to reduce blood pressure.

Metoprolol/HCT is a beta-blocker that works by blocking the effects of the hormone epinephrine, and also has a diuretic effect to reduce blood pressure. Used for Hypertension, Atrial fibrillation for stroke prevention.

Likelihood of approval
56.3% vs 58.3% industry baseline
If approved by FDA: likely 2028–2030
Steps remaining: NDA/BLA submission
Confidence: High
Why this estimate
  • Baseline phase 3 → approval rate +58.3pp
    Industry-wide phase 3 drugs reach approval ~58.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
  • Cardiovascular Phase 3 risk -2.0pp
    Modern cardiovascular outcome trials are large + long; many fail to beat aggressive standard-of-care.
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2028–2030
EMA EU 2029–2031 +0.7 yr
MHRA GB 2029–2031 +0.7 yr
Health Canada CA 2029–2032 +0.9 yr
TGA AU 2029–2032 +1.2 yr
PMDA JP 2029–2032 +1.5 yr
NMPA CN 2030–2033 +2.3 yr
MFDS KR 2029–2032 +1.4 yr
CDSCO IN 2029–2033 +1.8 yr
ANVISA BR 2030–2033 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic namemetoprolol/HCT
SponsorAbbott
Drug classBeta-blocker
TargetBeta-1 adrenergic receptor
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

By blocking the effects of epinephrine, metoprolol/HCT reduces the heart rate and blood pressure, which can help to prevent heart failure and reduce the risk of stroke. The diuretic effect of HCT helps to remove excess fluid from the body, further reducing blood pressure.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about metoprolol/HCT

What is metoprolol/HCT?

metoprolol/HCT is a Beta-blocker drug developed by Abbott, indicated for Hypertension, Atrial fibrillation for stroke prevention.

How does metoprolol/HCT work?

Metoprolol/HCT is a beta-blocker that works by blocking the effects of the hormone epinephrine, and also has a diuretic effect to reduce blood pressure.

What is metoprolol/HCT used for?

metoprolol/HCT is indicated for Hypertension, Atrial fibrillation for stroke prevention.

Who makes metoprolol/HCT?

metoprolol/HCT is developed by Abbott (see full Abbott pipeline at /company/abbott).

What drug class is metoprolol/HCT in?

metoprolol/HCT belongs to the Beta-blocker class. See all Beta-blocker drugs at /class/beta-blocker.

What development phase is metoprolol/HCT in?

metoprolol/HCT is in Phase 3.

What are the side effects of metoprolol/HCT?

Common side effects of metoprolol/HCT include Dizziness, Fatigue, Headache, Nausea, Diarrhea.

What does metoprolol/HCT target?

metoprolol/HCT targets Beta-1 adrenergic receptor and is a Beta-blocker.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing