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Inhaled Nitric Oxide (iNO) use

The University of Texas Health Science Center, Houston · FDA-approved active Small molecule

Inhaled nitric oxide is a vasodilator gas that selectively relaxes pulmonary vascular smooth muscle to reduce pulmonary hypertension and improve oxygenation.

Inhaled nitric oxide is a vasodilator gas that selectively relaxes pulmonary vascular smooth muscle to reduce pulmonary hypertension and improve oxygenation. Used for Persistent pulmonary hypertension of the newborn (PPHN), Acute respiratory distress syndrome (ARDS) with pulmonary hypertension, Pulmonary hypertension associated with left heart disease.

At a glance

Generic nameInhaled Nitric Oxide (iNO) use
SponsorThe University of Texas Health Science Center, Houston
Drug classInhaled vasodilator
TargetSoluble guanylate cyclase
ModalitySmall molecule
Therapeutic areaPulmonary/Cardiovascular
PhaseFDA-approved

Mechanism of action

Nitric oxide (NO) is an endogenous signaling molecule that activates guanylate cyclase in vascular smooth muscle cells, increasing cyclic GMP levels and causing vasodilation. When inhaled, NO preferentially dilates pulmonary vessels while being rapidly inactivated by hemoglobin in the systemic circulation, allowing selective pulmonary vasodilation without systemic hypotension. This reduces right ventricular afterload and improves ventilation-perfusion matching in the lungs.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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