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Caprelsa (VANDETANIB)

Sanofi · FDA-approved approved Small molecule Quality 64/100

Caprelsa works by blocking a specific protein that helps cancer cells grow and spread.

At a glance

Generic nameVANDETANIB
SponsorSanofi
Drug classKinase Inhibitor [EPC]
TargetReceptor-interacting serine/threonine-protein kinase 2
ModalitySmall molecule
Therapeutic areaOncology
PhaseFDA-approved
First approval2011

Mechanism of action

In vitro studies have shown that vandetanib inhibits the tyrosine kinase activity of the EGFR and VEGFR families, RET, BRK, TIE2, and members of the EPH receptor and Src kinase families. These receptor tyrosine kinases are involved in both normal cellular function and pathologic processes such as oncogenesis, metastasis, tumor angiogenesis, and maintenance of the tumor microenvironment. In addition, the N-desmethyl metabolite of the drug, representing to 17.1% of vandetanib exposure, has similar inhibitory activity to the parent compound for VEGF receptors (KDR and Flt-1) and EGFR. In vitro, vandetanib inhibited epidermal growth factor (EGF)-stimulated receptor tyrosine kinase phosphorylation in tumor cells and endothelial cells and VEGF-stimulated tyrosine kinase phosphorylation in endothelial cells. In vivo, vandetanib administration reduced tumor cell-induced angiogenesis, tumor vessel permeability, and inhibited tumor growth and metastasis in mouse models

Approved indications

Boxed warnings

Common side effects

Drug interactions

Key clinical trials

Patents

PatentExpiryType

Primary sources

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SourceUsed for
FDA labelMechanism, indications, dosing, boxed warnings, drug interactions
ClinicalTrials.govTrial enrolment, design, endpoints, results
FDA Orange BookPatents + exclusivity

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