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Tranexamic Acid (TXA)

NYU Langone Health · FDA-approved active Small molecule

Tranexamic acid inhibits the breakdown of blood clots by blocking plasminogen activation, thereby reducing fibrinolysis and promoting clot stability.

Tranexamic acid inhibits the breakdown of blood clots by blocking plasminogen activation, thereby reducing fibrinolysis and promoting clot stability. Used for Excessive bleeding or hemorrhage (perioperative and trauma settings), Menorrhagia (heavy menstrual bleeding), Hemophilia and other bleeding disorders.

At a glance

Generic nameTranexamic Acid (TXA)
Also known asTXA, Lysteda, Cyklokapron, Tranexamic acid, nebulization
SponsorNYU Langone Health
Drug classAntifibrinolytic agent
TargetPlasminogen / Plasmin
ModalitySmall molecule
Therapeutic areaHematology / Hemostasis
PhaseFDA-approved

Mechanism of action

Tranexamic acid is a competitive inhibitor of plasminogen and plasmin, enzymes responsible for dissolving fibrin in blood clots. By preventing the conversion of plasminogen to plasmin, it reduces fibrinolysis (clot breakdown) and helps maintain hemostasis. This mechanism makes it useful in conditions characterized by excessive bleeding or fibrinolysis.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results