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NSAID (Ketorolac/Ibuprofen)

Thomas Jefferson University · FDA-approved active Small molecule

NSAIDs inhibit cyclooxygenase (COX) enzymes to reduce prostaglandin synthesis, thereby decreasing inflammation, pain, and fever.

NSAIDs inhibit cyclooxygenase (COX) enzymes to reduce prostaglandin synthesis, thereby decreasing inflammation, pain, and fever. Used for Acute pain (ketorolac), Mild to moderate pain and inflammation (ibuprofen), Fever reduction.

At a glance

Generic nameNSAID (Ketorolac/Ibuprofen)
SponsorThomas Jefferson University
Drug classNon-steroidal anti-inflammatory drug (NSAID)
TargetCOX-1 and COX-2
ModalitySmall molecule
Therapeutic areaPain Management, Inflammation
PhaseFDA-approved

Mechanism of action

Ketorolac and ibuprofen are non-selective COX inhibitors that block both COX-1 and COX-2 enzymes. By reducing prostaglandin production, they suppress inflammatory mediators and provide analgesic and antipyretic effects. Ketorolac is a potent short-acting NSAID typically used for acute moderate-to-severe pain, while ibuprofen is a milder, longer-acting NSAID used for mild-to-moderate pain and inflammation.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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