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n-acetylcystine + albuterol

University of California, San Francisco · FDA-approved active Small molecule

N-acetylcysteine reduces mucus viscosity by breaking disulfide bonds in mucoproteins, while albuterol bronchodilates airways via beta-2 adrenergic stimulation, together improving airway clearance and breathing.

N-acetylcysteine reduces mucus viscosity by breaking disulfide bonds in mucoproteins, while albuterol bronchodilates airways via beta-2 adrenergic stimulation, together improving airway clearance and breathing. Used for Cystic fibrosis, Chronic obstructive pulmonary disease with excessive mucus production, Bronchiectasis.

At a glance

Generic namen-acetylcystine + albuterol
Also known asMucomyst, NAC, n-acetylcystine
SponsorUniversity of California, San Francisco
Drug classMucolytic + bronchodilator combination
TargetBeta-2 adrenergic receptor (albuterol); disulfide bonds in mucoproteins (N-acetylcysteine)
ModalitySmall molecule
Therapeutic areaRespiratory
PhaseFDA-approved

Mechanism of action

N-acetylcysteine acts as a mucolytic agent that cleaves mucus glycoproteins, reducing sputum thickness and improving clearance. Albuterol is a short-acting beta-2 agonist that relaxes bronchial smooth muscle, dilating airways. The combination enhances both mucus mobilization and airway patency, facilitating expectoration and gas exchange.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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