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N-acetylcystein

Assistance Publique - Hôpitaux de Paris · FDA-approved active Small molecule

N-acetylcysteine (NAC) replenishes intracellular glutathione and acts as a free radical scavenger to reduce oxidative stress and cellular damage.

N-acetylcysteine (NAC) replenishes intracellular glutathione and acts as a free radical scavenger to reduce oxidative stress and cellular damage. Used for Chronic obstructive pulmonary disease (COPD) with chronic bronchitis, Acetaminophen (paracetamol) overdose and toxicity, Acute respiratory distress syndrome (ARDS).

At a glance

Generic nameN-acetylcystein
Also known asFluimucil (Zambon, Italy), ACC (Hexal AG), Acemuc (Betapharm, Germany), Acetyst (Ristert, Germany), Acetadote (Cumberland Pharmaceuticals)
SponsorAssistance Publique - Hôpitaux de Paris
Drug classAntioxidant; mucolytic agent
TargetGlutathione synthesis pathway; free radicals; mucus proteins
ModalitySmall molecule
Therapeutic areaRespiratory; Toxicology; General supportive care
PhaseFDA-approved

Mechanism of action

NAC is a prodrug that is rapidly deacetylated to cysteine, which serves as a precursor for glutathione synthesis—the body's primary intracellular antioxidant. By restoring glutathione levels and directly scavenging reactive oxygen species, NAC protects cells from oxidative injury and supports detoxification pathways. It also has mucolytic properties that help break down mucus viscosity.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results