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JNJ16269110

Johnson & Johnson Pharmaceutical Research & Development, L.L.C. · Phase 2 active Small molecule

JNJ16269110 is a Glucagon receptor antagonist Small molecule drug developed by Johnson & Johnson Pharmaceutical Research & Development, L.L.C.. It is currently in Phase 2 development for Type 2 diabetes.

JNJ16269110 is a selective and potent antagonist of the human glucagon receptor.

JNJ16269110 is a selective and potent antagonist of the human glucagon receptor. Used for Type 2 diabetes.

Likelihood of approval
18.3% vs 15.3% industry baseline
If approved by FDA: likely 2031–2034
Steps remaining: Phase 3 → NDA/BLA submission
Confidence: Medium
Why this estimate
  • Baseline phase 2 → approval rate +15.3pp
    Industry-wide phase 2 drugs reach approval ~15.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
  • Big-pharma sponsor +3.0pp
    Johnson & Johnson Pharmaceutical Research & Development, L.L.C. is a top-20 pharma sponsor — historical approval rates run ~3pp above average due to scale, regulatory experience, and trial-design quality.
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2031–2034
EMA EU 2032–2035 +0.7 yr
MHRA GB 2032–2035 +0.7 yr
Health Canada CA 2032–2036 +0.9 yr
TGA AU 2032–2036 +1.2 yr
PMDA JP 2032–2036 +1.5 yr
NMPA CN 2033–2037 +2.3 yr
MFDS KR 2032–2036 +1.4 yr
CDSCO IN 2032–2037 +1.8 yr
ANVISA BR 2033–2037 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic nameJNJ16269110
SponsorJohnson & Johnson Pharmaceutical Research & Development, L.L.C.
Drug classGlucagon receptor antagonist
TargetGlucagon receptor
ModalitySmall molecule
Therapeutic areaDiabetes
PhasePhase 2

Mechanism of action

Glucagon is a hormone that raises blood glucose levels by stimulating glycogenolysis and gluconeogenesis. By blocking glucagon receptors, JNJ16269110 reduces blood glucose levels, making it a potential treatment for type 2 diabetes.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about JNJ16269110

What is JNJ16269110?

JNJ16269110 is a Glucagon receptor antagonist drug developed by Johnson & Johnson Pharmaceutical Research & Development, L.L.C., indicated for Type 2 diabetes.

How does JNJ16269110 work?

JNJ16269110 is a selective and potent antagonist of the human glucagon receptor.

What is JNJ16269110 used for?

JNJ16269110 is indicated for Type 2 diabetes.

Who makes JNJ16269110?

JNJ16269110 is developed by Johnson & Johnson Pharmaceutical Research & Development, L.L.C. (see full Johnson & Johnson Pharmaceutical Research & Development, L.L.C. pipeline at /company/johnson-johnson-pharmaceutical-research-development-l-l-c).

What drug class is JNJ16269110 in?

JNJ16269110 belongs to the Glucagon receptor antagonist class. See all Glucagon receptor antagonist drugs at /class/glucagon-receptor-antagonist.

What development phase is JNJ16269110 in?

JNJ16269110 is in Phase 2.

What are the side effects of JNJ16269110?

Common side effects of JNJ16269110 include Nausea, Diarrhea, Abdominal pain.

What does JNJ16269110 target?

JNJ16269110 targets Glucagon receptor and is a Glucagon receptor antagonist.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing