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Ivacaftor+lumacaftor

Assistance Publique - Hôpitaux de Paris · FDA-approved active Small molecule

Lumacaftor corrects the trafficking of defective CFTR protein to the cell surface, while ivacaftor enhances the channel activity of CFTR, together restoring chloride transport in cystic fibrosis.

Lumacaftor corrects the trafficking of defective CFTR protein to the cell surface, while ivacaftor enhances the channel activity of CFTR, together restoring chloride transport in cystic fibrosis. Used for Cystic fibrosis in patients homozygous for the F508del-CFTR mutation or heterozygous for F508del and a minimal function mutation.

At a glance

Generic nameIvacaftor+lumacaftor
Also known asOrkambi
SponsorAssistance Publique - Hôpitaux de Paris
Drug classCFTR modulator combination (corrector + potentiator)
TargetCFTR (cystic fibrosis transmembrane conductance regulator)
ModalitySmall molecule
Therapeutic areaRespiratory / Genetic disease
PhaseFDA-approved

Mechanism of action

Lumacaftor is a CFTR corrector that helps misfolded CFTR protein (particularly the F508del mutation) fold correctly and traffic to the apical membrane. Ivacaftor is a CFTR potentiator that increases the open probability and duration of the CFTR channel at the cell surface. Together, they address both the trafficking defect and the gating defect of mutant CFTR, improving ion and fluid transport in the airways.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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