Last reviewed · How we verify

Intra-Arterial TNK

Tianjin Huanhu Hospital · Phase 3 active Small molecule

TNK (tenecteplase) is a fibrinolytic agent that dissolves blood clots by converting plasminogen to plasmin when administered directly into an artery.

TNK (tenecteplase) is a fibrinolytic agent that dissolves blood clots by converting plasminogen to plasmin when administered directly into an artery. Used for Acute ischemic stroke (intra-arterial administration).

At a glance

Generic nameIntra-Arterial TNK
SponsorTianjin Huanhu Hospital
Drug classFibrinolytic agent / Thrombolytic
TargetPlasminogen / Fibrin
ModalitySmall molecule
Therapeutic areaCardiovascular / Neurology
PhasePhase 3

Mechanism of action

TNK is a genetically engineered variant of tissue plasminogen activator (tPA) with enhanced fibrin specificity and longer half-life. When delivered intra-arterially, it directly activates the fibrinolytic cascade at the site of thrombosis, promoting rapid clot dissolution. This approach is used in acute ischemic stroke to restore blood flow to affected brain tissue.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: