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ERA as specific PAH treatment

Eli Lilly and Company · Phase 3 active Small molecule

Endothelin receptor antagonists (ERAs) block endothelin-1 signaling in pulmonary vasculature to reduce vasoconstriction and improve hemodynamics in pulmonary arterial hypertension.

Endothelin receptor antagonists (ERAs) block endothelin-1 signaling in pulmonary vasculature to reduce vasoconstriction and improve hemodynamics in pulmonary arterial hypertension. Used for Pulmonary arterial hypertension (PAH).

At a glance

Generic nameERA as specific PAH treatment
SponsorEli Lilly and Company
Drug classEndothelin receptor antagonist
TargetEndothelin receptor (ETA/ETB)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

ERAs inhibit the binding of endothelin-1 to endothelin receptors (ETA and/or ETB) on vascular smooth muscle cells and endothelial cells. Endothelin-1 is a potent vasoconstrictor and mitogen implicated in PAH pathophysiology. By antagonizing these receptors, ERAs reduce pulmonary vascular resistance, improve right ventricular function, and delay disease progression in PAH patients.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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