Last reviewed · How we verify

CsA+MMF+CS

Pfizer · FDA-approved active Small molecule

This combination suppresses the immune system through multiple pathways: cyclosporine inhibits T-cell activation, mycophenolate mofetil blocks lymphocyte proliferation, and corticosteroids broadly dampen inflammatory responses.

This combination suppresses the immune system through multiple pathways: cyclosporine inhibits T-cell activation, mycophenolate mofetil blocks lymphocyte proliferation, and corticosteroids broadly dampen inflammatory responses. Used for Prevention of organ rejection in renal transplant recipients, Prevention of organ rejection in cardiac transplant recipients, Prevention of organ rejection in hepatic transplant recipients.

At a glance

Generic nameCsA+MMF+CS
SponsorPfizer
Drug classImmunosuppressive combination therapy
TargetCalcineurin (CsA); IMPDH type II (MMF); glucocorticoid receptor (CS)
ModalitySmall molecule
Therapeutic areaImmunology / Transplantation
PhaseFDA-approved

Mechanism of action

Cyclosporine (CsA) binds calcineurin to prevent T-cell receptor signaling and IL-2 production. Mycophenolate mofetil (MMF) selectively inhibits inosine monophosphate dehydrogenase, depleting guanosine nucleotides needed for lymphocyte proliferation. Corticosteroids (CS) reduce inflammatory cytokine production and immune cell trafficking. Together, these agents provide synergistic immunosuppression for transplant rejection prevention.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: