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botulinum toxin neuromodulator

Galderma R&D · Phase 3 active Biologic

Botulinum toxin blocks acetylcholine release at the neuromuscular junction by cleaving SNARE proteins, causing temporary muscle paralysis.

Botulinum toxin blocks acetylcholine release at the neuromuscular junction by cleaving SNARE proteins, causing temporary muscle paralysis. Used for Chronic migraine, Cervical dystonia, Blepharospasm.

At a glance

Generic namebotulinum toxin neuromodulator
Also known asQM1114-DP
SponsorGalderma R&D
Drug classNeuromuscular blocking agent
TargetSNARE complex (synaptobrevin, SNAP-25, syntaxin)
ModalityBiologic
Therapeutic areaNeurology, Aesthetics, Pain Management
PhasePhase 3

Mechanism of action

Botulinum toxin is a neurotoxin that irreversibly cleaves proteins required for acetylcholine vesicle fusion and release at the presynaptic terminal. This prevents neuromuscular transmission, resulting in temporary flaccid paralysis of targeted muscles. The effect is reversible over months as the body regenerates new neuromuscular junctions.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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