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BOTOX®, Allergan

Assistance Publique - Hôpitaux de Paris · Phase 3 active Small molecule

BOTOX blocks the release of acetylcholine at the neuromuscular junction by cleaving SNARE proteins, causing temporary paralysis of muscles.

BOTOX blocks the release of acetylcholine at the neuromuscular junction by cleaving SNARE proteins, causing temporary paralysis of muscles. Used for Cervical dystonia, Blepharospasm, Strabismus.

At a glance

Generic nameBOTOX®, Allergan
Also known asBOTOX
SponsorAssistance Publique - Hôpitaux de Paris
Drug classBotulinum toxin type A
TargetSNARE proteins (specifically SNAP-25)
ModalitySmall molecule
Therapeutic areaNeurology, Dermatology, Ophthalmology
PhasePhase 3

Mechanism of action

Botulinum toxin type A binds to presynaptic nerve terminals and cleaves SNARE (soluble N-ethylmaleimide-sensitive factor attachment protein receptor) proteins, preventing acetylcholine vesicles from fusing with the cell membrane. This results in temporary muscle paralysis lasting 3–4 months. The effect is reversible as the body gradually regenerates new nerve terminals.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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