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NCT06334900: Ata-GAD

Retrospective Analysis of the French National Cohort of Patients With GAD Antibodies and Cerebellar Ataxia

Status unknown Last updated 28 March 2024
What this trial tests

trial testing GAD patients in GAD-receptor Antibodies-associated Encephalitis in 100 participants. Status unknown.

Timeline
1 October 2023
Primary endpoint
1 January 2024
30 June 2024

Quick facts

Lead sponsorHospices Civils de Lyon
StatusStatus unknown
Study typeOBSERVATIONAL
Enrollment100
Start date1 October 2023
Primary completion1 January 2024
Estimated completion30 June 2024
Sites1 location across France

Drugs / interventions tested

Conditions studied

Sponsor

Hospices Civils de Lyon — full company profile →

Who can join

18 and older, any sex, with GAD-receptor Antibodies-associated Encephalitis. Patients with the condition only — healthy volunteers not accepted.

Sponsor's own description

Glutamic acid decarboxylase (GAD) is an enzyme whose function in the body is to decarboxylate glutamate to GABA. GAD65 antibodies (GAD65Ab) have been associated with type-1 diabetes (80% of new-onset patients) and various neurological conditions, mainly stiff-person syndrome (SPS/PERM), cerebellar ataxia (CA), limbic encephalitis (LE) and temporal lobe epilepsy. These syndromes all seem to result from a reduced transmission of GABA. These neurological conditions are rare and can cause symptoms like confusion, memory loss, muscle stiffness, muscle spasms, behavioural disorders, and pharmacoresistant epilepsy. When finding high levels of GAD65-Ab in the serum, a cerebrospinal fluid (CSF) sample should be taken to look for oligoclonal IgG bands and intrathecal GAD-Ab production to prove an auto-immune cause for the various neurological symptoms.

Publications & conference data

No peer-reviewed publications indexed yet for this trial.

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Other Hospices Civils de Lyon trials

Trials by the same sponsor.

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Data sources for this page

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