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NCT03444753

An Investigational Immunotherapy Study of BMS-986299 Alone and in Combination With Nivolumab and Ipilimumab in Participants With Solid Cancers That Have Spread or Cannot be Removed

Terminated Phase 1 Last updated 23 May 2022
What this trial tests

Phase 1 trial testing BMS-986299 in Advanced Cancer in 82 participants. Terminated before completion.

Timeline
5 April 2018
Primary endpoint
14 February 2022
14 February 2022

Quick facts

Lead sponsorBristol-Myers Squibb
PhasePhase 1
StatusTerminated
Study typeINTERVENTIONAL
Allocationnon randomized
Designparallel
Maskingnone
Primary purposetreatment
Enrollment82
Start date5 April 2018
Primary completion14 February 2022
Estimated completion14 February 2022
Sites9 locations across United States

Drugs / interventions tested

Conditions studied

Sponsor

Bristol-Myers Squibb — full company profile →

Who can join

18 and older, any sex, with Advanced Cancer. Patients with the condition only — healthy volunteers not accepted.

Sponsor's own description

The purpose of this study is to determine whether BMS-986299 both by itself and in combination with Nivolumab and Ipilimumab is safe and tolerable in the treatment of advanced solid tumors. In addition, the ability of study drugs to stimulate an immune response against cancer will be investigated.

Publications & conference data

8 peer-reviewed publications reference this trial (live from Europe PMC):

  1. Therapeutic modulation of inflammasome pathways.
    Chauhan D, Vande Walle L, Lamkanfi M. · · 2020 · cited 163× · PMID 32770571 · DOI 10.1111/imr.12908
  2. Harnessing innate immune pathways for therapeutic advancement in cancer.
    Hu A, Sun L, Lin H, Liao Y, et al · · 2024 · cited 150× · PMID 38523155 · DOI 10.1038/s41392-024-01765-9
  3. NLRP3 inflammasome-mediated cytokine production and pyroptosis cell death in breast cancer.
    Faria SS, Costantini S, de Lima VCC, de Andrade VP, et al · · 2021 · cited 144× · PMID 33840390 · DOI 10.1186/s12929-021-00724-8
  4. IL-1 and autoinflammatory disease: biology, pathogenesis and therapeutic targeting.
    Broderick L, Hoffman HM. · · 2022 · cited 112× · PMID 35729334 · DOI 10.1038/s41584-022-00797-1
  5. Targeting Innate Immunity in Cancer Therapy.
    Rameshbabu S, Labadie BW, Argulian A, Patnaik A. · · 2021 · cited 90× · PMID 33572196 · DOI 10.3390/vaccines9020138
  6. NLRP3 inflammasome: a key player in the pathogenesis of life-style disorders.
    Ramachandran R, Manan A, Kim J, Choi S. · · 2024 · cited 67× · PMID 38945951 · DOI 10.1038/s12276-024-01261-8
  7. Involvement of inflammasomes in tumor microenvironment and tumor therapies.
    Zhang Z, Li X, Wang Y, Wei Y, et al · · 2023 · cited 55× · PMID 36932407 · DOI 10.1186/s13045-023-01407-7
  8. The discovery of NLRP3 and its function in cryopyrin-associated periodic syndromes and innate immunity.
    Putnam CD, Broderick L, Hoffman HM. · · 2024 · cited 27× · PMID 38146057 · DOI 10.1111/imr.13292

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Data sources for this page

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