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Xalatan+Cosopt

University of Turin, Italy · FDA-approved active Small molecule ✓ Verified May 2026

Xalatan+Cosopt is a Prostaglandin analog + carbonic anhydrase inhibitor + beta-blocker combination Small molecule drug developed by University of Turin, Italy. It is currently FDA-approved for Open-angle glaucoma, Ocular hypertension.

This combination reduces intraocular pressure by combining a prostaglandin analog (latanoprost) that increases uveoscleral outflow with a carbonic anhydrase inhibitor and beta-blocker (dorzolamide/timolol) that decrease aqueous humor production.

Xalatan (latanoprost 0.005%) and Cosopt (dorzolamide 2%/timolol maleate 0.5% fixed combination) are medications used to treat conditions such as open-angle glaucoma and ocular hypertension. They are small molecule drugs that work through different mechanisms, with Xalatan likely acting as a prostaglandin analog and Cosopt acting as a carbonic anhydrase inhibitor and beta-adrenergic receptor blocker.

At a glance

Generic nameXalatan+Cosopt
SponsorUniversity of Turin, Italy
Drug classProstaglandin analog + carbonic anhydrase inhibitor + beta-blocker combination
TargetProstaglandin F receptor (FP receptor); carbonic anhydrase II; beta-adrenergic receptors
ModalitySmall molecule
Therapeutic areaOphthalmology
PhaseFDA-approved

Mechanism of action

Xalatan (latanoprost) is a prostaglandin F analog that enhances uveoscleral drainage of aqueous humor. Cosopt (dorzolamide/timolol) combines dorzolamide, a topical carbonic anhydrase inhibitor that reduces aqueous humor secretion, with timolol, a non-selective beta-blocker that also decreases aqueous humor production. Together, these agents work synergistically through multiple mechanisms to lower intraocular pressure.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

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Frequently asked questions about Xalatan+Cosopt

What is Xalatan+Cosopt?

Xalatan+Cosopt is a Prostaglandin analog + carbonic anhydrase inhibitor + beta-blocker combination drug developed by University of Turin, Italy, indicated for Open-angle glaucoma, Ocular hypertension.

How does Xalatan+Cosopt work?

This combination reduces intraocular pressure by combining a prostaglandin analog (latanoprost) that increases uveoscleral outflow with a carbonic anhydrase inhibitor and beta-blocker (dorzolamide/timolol) that decrease aqueous humor production.

What is Xalatan+Cosopt used for?

Xalatan+Cosopt is indicated for Open-angle glaucoma, Ocular hypertension.

Who makes Xalatan+Cosopt?

Xalatan+Cosopt is developed and marketed by University of Turin, Italy (see full University of Turin, Italy pipeline at /company/university-of-turin-italy).

What drug class is Xalatan+Cosopt in?

Xalatan+Cosopt belongs to the Prostaglandin analog + carbonic anhydrase inhibitor + beta-blocker combination class. See all Prostaglandin analog + carbonic anhydrase inhibitor + beta-blocker combination drugs at /class/prostaglandin-analog-carbonic-anhydrase-inhibitor-beta-blocker-combination.

What development phase is Xalatan+Cosopt in?

Xalatan+Cosopt is FDA-approved (marketed).

What are the side effects of Xalatan+Cosopt?

Common side effects of Xalatan+Cosopt include Conjunctival hyperemia, Eye irritation/discomfort, Increased iris pigmentation, Eyelash growth, Ocular surface disease, Headache.

What does Xalatan+Cosopt target?

Xalatan+Cosopt targets Prostaglandin F receptor (FP receptor); carbonic anhydrase II; beta-adrenergic receptors and is a Prostaglandin analog + carbonic anhydrase inhibitor + beta-blocker combination.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing