Last reviewed · How we verify

TNK-tPA

Beijing Tiantan Hospital · Phase 3 active Small molecule

TNK-tPA is a genetically engineered tissue plasminogen activator that converts plasminogen to plasmin to dissolve blood clots.

TNK-tPA is a genetically engineered tissue plasminogen activator that converts plasminogen to plasmin to dissolve blood clots. Used for Acute myocardial infarction (AMI), Acute ischemic stroke.

At a glance

Generic nameTNK-tPA
Also known asrtPA
SponsorBeijing Tiantan Hospital
Drug classFibrinolytic agent / Thrombolytic
TargetPlasminogen / Fibrin
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

TNK-tPA (tenecteplase) is a fibrinolytic agent derived from natural tissue plasminogen activator (tPA) with amino acid substitutions that improve its pharmacokinetic properties. It binds to fibrin in thrombi and catalyzes the conversion of plasminogen to plasmin, which degrades the fibrin matrix and dissolves the clot. The modifications make it more fibrin-specific and resistant to plasminogen activator inhibitor-1 compared to wild-type tPA, allowing for faster administration and improved efficacy.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: