Last reviewed · How we verify

Tacrolimus (Arm A)

Weill Medical College of Cornell University · FDA-approved active Small molecule Under review

Tacrolimus (Arm A) is a Calcineurin inhibitor Small molecule drug developed by Weill Medical College of Cornell University. It is currently FDA-approved for Organ transplant rejection prophylaxis (kidney, heart, liver, pancreas), Atopic dermatitis, Autoimmune uveitis. Also known as: Prograf.

Tacrolimus inhibits calcineurin phosphatase, blocking T-cell activation and proliferation by preventing transcription of IL-2 and other cytokine genes.

Tacrolimus is a small molecule inhibitor of the FK506-binding protein 1A, which is involved in the regulation of the immune system. It is used to treat conditions such as Graft vs Host Disease, Malignancy, Immunosuppression, Kidney Transplantation, and Liver Transplantation.

At a glance

Generic nameTacrolimus (Arm A)
Also known asPrograf
SponsorWeill Medical College of Cornell University
Drug classCalcineurin inhibitor
TargetCalcineurin (via FKBP12 binding)
ModalitySmall molecule
Therapeutic areaImmunology
PhaseFDA-approved

Mechanism of action

Tacrolimus binds to the immunophilin FKBP12, and this complex inhibits calcineurin, a serine/threonine phosphatase essential for dephosphorylating NFAT (nuclear factor of activated T cells). By preventing NFAT nuclear translocation, tacrolimus suppresses the transcription of interleukin-2 and other T-cell activation genes, resulting in potent immunosuppression. This mechanism makes it effective for preventing organ rejection and treating autoimmune conditions.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about Tacrolimus (Arm A)

What is Tacrolimus (Arm A)?

Tacrolimus (Arm A) is a Calcineurin inhibitor drug developed by Weill Medical College of Cornell University, indicated for Organ transplant rejection prophylaxis (kidney, heart, liver, pancreas), Atopic dermatitis, Autoimmune uveitis.

How does Tacrolimus (Arm A) work?

Tacrolimus inhibits calcineurin phosphatase, blocking T-cell activation and proliferation by preventing transcription of IL-2 and other cytokine genes.

What is Tacrolimus (Arm A) used for?

Tacrolimus (Arm A) is indicated for Organ transplant rejection prophylaxis (kidney, heart, liver, pancreas), Atopic dermatitis, Autoimmune uveitis, Autoimmune hepatitis.

Who makes Tacrolimus (Arm A)?

Tacrolimus (Arm A) is developed and marketed by Weill Medical College of Cornell University (see full Weill Medical College of Cornell University pipeline at /company/weill-medical-college-of-cornell-university).

Is Tacrolimus (Arm A) also known as anything else?

Tacrolimus (Arm A) is also known as Prograf.

What drug class is Tacrolimus (Arm A) in?

Tacrolimus (Arm A) belongs to the Calcineurin inhibitor class. See all Calcineurin inhibitor drugs at /class/calcineurin-inhibitor.

What development phase is Tacrolimus (Arm A) in?

Tacrolimus (Arm A) is FDA-approved (marketed).

What are the side effects of Tacrolimus (Arm A)?

Common side effects of Tacrolimus (Arm A) include Nephrotoxicity, Hypertension, Hyperglycemia, Tremor, Headache, Infection.

What does Tacrolimus (Arm A) target?

Tacrolimus (Arm A) targets Calcineurin (via FKBP12 binding) and is a Calcineurin inhibitor.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing