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Prandin (repaglinide)

Novo Nordisk · FDA-approved active Verified Quality 80/100

Repaglinide lowers blood glucose levels by stimulating insulin release from the pancreas through ATP-dependent potassium channel blockade.

At a glance

Generic namerepaglinide
SponsorNovo Nordisk
Drug classGlinide [EPC]
TargetATP-binding cassette sub-family C member 8
Therapeutic areaMetabolic
PhaseFDA-approved
First approval1997

Mechanism of action

Repaglinide works by binding to characterizable sites on ATP-dependent potassium channels in the beta cell membrane. This binding causes the potassium channels to close, leading to depolarization of the beta cell. The depolarization opens calcium channels, allowing an influx of calcium ions into the cell. The increased calcium levels trigger the release of insulin from the beta cell. This action is glucose-dependent, meaning that insulin release is only stimulated when glucose levels are high. At low glucose concentrations, insulin release is diminished.

Approved indications

Common side effects

Drug interactions

Key clinical trials

Primary sources

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SourceUsed for
FDA labelMechanism, indications, dosing, boxed warnings, drug interactions
ClinicalTrials.govTrial enrolment, design, endpoints, results

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