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Prednisone and Belimumab

Peking Union Medical College Hospital · FDA-approved active Small molecule

Prednisone suppresses immune responses through glucocorticoid receptor activation, while belimumab inhibits B-lymphocyte stimulator (BLyS) to reduce autoreactive B cell survival and antibody production.

Prednisone suppresses immune responses through glucocorticoid receptor activation, while belimumab inhibits B-lymphocyte stimulator (BLyS) to reduce autoreactive B cell survival and antibody production. Used for Systemic lupus erythematosus (SLE).

At a glance

Generic namePrednisone and Belimumab
Also known asBelimumab treatment
SponsorPeking Union Medical College Hospital
Drug classCorticosteroid + Monoclonal antibody (BLyS inhibitor)
TargetGlucocorticoid receptor (prednisone); BLyS/BAFF (belimumab)
ModalitySmall molecule
Therapeutic areaImmunology
PhaseFDA-approved

Mechanism of action

Prednisone is a corticosteroid that broadly dampens inflammatory and immune responses by binding glucocorticoid receptors and suppressing cytokine production and immune cell activation. Belimumab is a monoclonal antibody that specifically targets BLyS (also called BAFF), a key survival factor for B cells, thereby reducing pathogenic autoantibody-producing B cells. Together, this combination provides both broad immunosuppression and targeted B cell depletion.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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