Last reviewed · How we verify

Placebo to interferon beta-1a

Celgene · Phase 3 active Small molecule

Interferon beta-1a activates interferon signaling pathways to modulate immune responses and reduce inflammatory activity.

Interferon beta-1a activates interferon signaling pathways to modulate immune responses and reduce inflammatory activity. Used for Relapsing-remitting multiple sclerosis, Secondary progressive multiple sclerosis.

At a glance

Generic namePlacebo to interferon beta-1a
SponsorCelgene
Drug classInterferon
TargetInterferon-beta receptor (IFNAR)
ModalitySmall molecule
Therapeutic areaImmunology
PhasePhase 3

Mechanism of action

Interferon beta-1a is a recombinant cytokine that binds to interferon-beta receptors on immune cells, triggering intracellular signaling cascades that enhance natural killer cell activity, increase antigen presentation, and promote anti-inflammatory cytokine production. This mechanism helps suppress autoreactive T cells and reduce demyelination in multiple sclerosis by shifting the immune response toward a more regulatory phenotype.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: