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PHENCYCLIDINE

Phase 2 active Small molecule

PHENCYCLIDINE is a phencyclidine drug. It is currently in Phase 2 development.

Phencyclidine blocks the action of glutamate at the NMDA receptor, disrupting normal neural communication.

Phencyclidine (PCP) is a small molecule drug that targets the glutamate NMDA receptor. It is a non-competitive antagonist, meaning it blocks the receptor's action without binding to the same site as the endogenous ligand. PCP is not FDA-approved for any indications and is primarily known for its dissociative and hallucinogenic effects. Its commercial status is unknown, and it is not widely available as a generic medication. Key safety considerations include its potential for abuse and dependence, as well as its ability to cause psychosis and other severe psychiatric symptoms.

Likelihood of approval
12.3% vs 15.3% industry baseline
If approved by FDA: likely 2031–2034
Steps remaining: Phase 3 → NDA/BLA submission
Confidence: Medium
Why this estimate
  • Baseline phase 2 → approval rate +15.3pp
    Industry-wide phase 2 drugs reach approval ~15.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
  • CNS / neurology attrition -3.0pp
    CNS drugs have historically high Phase 3 failure rates (notably in Alzheimer disease + major depression).
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2031–2034
EMA EU 2032–2035 +0.7 yr
MHRA GB 2032–2035 +0.7 yr
Health Canada CA 2032–2036 +0.9 yr
TGA AU 2032–2036 +1.2 yr
PMDA JP 2032–2036 +1.5 yr
NMPA CN 2033–2037 +2.3 yr
MFDS KR 2032–2036 +1.4 yr
CDSCO IN 2032–2037 +1.8 yr
ANVISA BR 2033–2037 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic namePHENCYCLIDINE
Drug classphencyclidine
TargetLysosomal Pro-X carboxypeptidase, Sigma non-opioid intracellular receptor 1, Glutamate NMDA receptor; GRIN1/GRIN2A
ModalitySmall molecule
Therapeutic areaNeuroscience
PhasePhase 2

Mechanism of action

Imagine your brain is a city with many streets (neurons) and intersections (synapses). Glutamate is like a traffic cop that helps guide the flow of information between streets. Phencyclidine is like a roadblock that prevents the traffic cop from doing its job, causing confusion and disrupting the normal flow of information.

Approved indications

No approved indications tracked.

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about PHENCYCLIDINE

What is PHENCYCLIDINE?

PHENCYCLIDINE is a phencyclidine drug.

How does PHENCYCLIDINE work?

Phencyclidine blocks the action of glutamate at the NMDA receptor, disrupting normal neural communication.

What drug class is PHENCYCLIDINE in?

PHENCYCLIDINE belongs to the phencyclidine class. See all phencyclidine drugs at /class/phencyclidine.

What development phase is PHENCYCLIDINE in?

PHENCYCLIDINE is in Phase 2.

What are the side effects of PHENCYCLIDINE?

Common side effects of PHENCYCLIDINE include Drug abuse, Toxicity to various agents.

What does PHENCYCLIDINE target?

PHENCYCLIDINE targets Lysosomal Pro-X carboxypeptidase, Sigma non-opioid intracellular receptor 1, Glutamate NMDA receptor; GRIN1/GRIN2A and is a phencyclidine.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing