Last reviewed · How we verify

Ovarian Function Suppression + Aromatase Inhibitor

NRG Oncology · Phase 3 active Small molecule

This combination suppresses ovarian estrogen production while blocking the conversion of androgens to estrogen, reducing total circulating estrogen in premenopausal women with hormone receptor-positive breast cancer.

This combination suppresses ovarian estrogen production while blocking the conversion of androgens to estrogen, reducing total circulating estrogen in premenopausal women with hormone receptor-positive breast cancer. Used for Hormone receptor-positive, HER2-negative breast cancer in premenopausal women (adjuvant or metastatic setting).

At a glance

Generic nameOvarian Function Suppression + Aromatase Inhibitor
SponsorNRG Oncology
Drug classCombination endocrine therapy (GnRH agonist/antagonist + aromatase inhibitor)
TargetGnRH receptor (ovarian suppression component) and aromatase enzyme (AI component)
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Ovarian function suppression (OFS) agents such as GnRH agonists or antagonists shut down the ovaries' production of estrogen in premenopausal women. Aromatase inhibitors (AIs) block the enzyme aromatase, which converts androgens to estrogen in peripheral tissues. Together, this dual approach maximizes estrogen deprivation in hormone receptor-positive breast cancer by eliminating both ovarian and peripheral sources of estrogen.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: