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Novel strategy Aspirin

Assistance Publique - Hôpitaux de Paris · FDA-approved active Small molecule

Aspirin irreversibly inhibits cyclooxygenase (COX) enzymes to reduce platelet aggregation and inflammation.

Aspirin irreversibly inhibits cyclooxygenase (COX) enzymes to reduce platelet aggregation and inflammation. Used for Cardiovascular disease prevention (secondary prevention post-myocardial infarction or stroke), Acute coronary syndrome, Mild to moderate pain and fever.

At a glance

Generic nameNovel strategy Aspirin
SponsorAssistance Publique - Hôpitaux de Paris
Drug classNonsteroidal anti-inflammatory drug (NSAID); antiplatelet agent
TargetCyclooxygenase-1 (COX-1); Cyclooxygenase-2 (COX-2)
ModalitySmall molecule
Therapeutic areaCardiovascular; Pain management; Inflammation
PhaseFDA-approved

Mechanism of action

Aspirin acetylates serine residues on COX-1 and COX-2, preventing the synthesis of prostaglandins and thromboxane A2. This reduces platelet activation and aggregation, lowering thrombotic risk, while also providing anti-inflammatory and analgesic effects through inhibition of prostaglandin-mediated inflammation.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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