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N Acetylcysteine A

Department of Medical Services Ministry of Public Health of Thailand · FDA-approved active Small molecule

N-acetylcysteine replenishes intracellular glutathione and acts as a mucolytic agent to reduce mucus viscosity and support antioxidant defenses.

N-acetylcysteine replenishes intracellular glutathione and acts as a mucolytic agent to reduce mucus viscosity and support antioxidant defenses. Used for Chronic bronchitis with thick secretions, Acetaminophen (paracetamol) overdose, Cystic fibrosis.

At a glance

Generic nameN Acetylcysteine A
SponsorDepartment of Medical Services Ministry of Public Health of Thailand
Drug classMucolytic agent / Antioxidant
TargetGlutathione synthesis pathway; mucus disulfide bonds
ModalitySmall molecule
Therapeutic areaRespiratory; Toxicology; General supportive care
PhaseFDA-approved

Mechanism of action

N-acetylcysteine (NAC) is a prodrug that is rapidly deacetylated to cysteine, which serves as a precursor for glutathione synthesis, the body's primary intracellular antioxidant. Additionally, NAC directly breaks disulfide bonds in mucoproteins, reducing mucus viscosity and improving clearance. These dual mechanisms make it useful in conditions characterized by oxidative stress, thick secretions, or acetaminophen toxicity.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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