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N-3 Polyunsaturated Fatty Acids

Sixth Affiliated Hospital, Sun Yat-sen University · Phase 3 active Small molecule

N-3 polyunsaturated fatty acids reduce triglycerides and modulate inflammatory pathways through incorporation into cell membranes and effects on eicosanoid metabolism.

N-3 polyunsaturated fatty acids reduce triglycerides and modulate inflammatory pathways through incorporation into cell membranes and effects on eicosanoid metabolism. Used for Hypertriglyceridemia, Cardiovascular disease prevention, Inflammation-related conditions.

At a glance

Generic nameN-3 Polyunsaturated Fatty Acids
Also known asn-3PUFA
SponsorSixth Affiliated Hospital, Sun Yat-sen University
Drug classOmega-3 fatty acid / Lipid-modifying agent
TargetGPR120, GPR40, PPAR-γ, LXR
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

N-3 PUFAs (primarily EPA and DHA) are incorporated into cell membranes where they displace arachidonic acid, reducing production of pro-inflammatory eicosanoids. They also activate G-protein coupled receptors and nuclear receptors (PPAR-γ, LXR) that suppress inflammatory gene expression and improve lipid metabolism, particularly reducing circulating triglycerides and modulating HDL/LDL ratios.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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