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Ivacaftor and Symdeko

University of Alabama at Birmingham · FDA-approved active Small molecule

Ivacaftor is a CFTR potentiator that increases the opening probability of the cystic fibrosis transmembrane conductance regulator (CFTR) channel, while Symdeko combines ivacaftor with lumacaftor, a CFTR corrector that helps misfolded CFTR protein reach the cell surface.

Ivacaftor is a CFTR potentiator that increases the opening probability of the cystic fibrosis transmembrane conductance regulator (CFTR) channel, while Symdeko combines ivacaftor with lumacaftor, a CFTR corrector that helps misfolded CFTR protein reach the cell surface. Used for Cystic fibrosis in patients with specific CFTR mutations (F508del and other gating mutations for ivacaftor; F508del-mediated CF for Symdeko).

At a glance

Generic nameIvacaftor and Symdeko
SponsorUniversity of Alabama at Birmingham
Drug classCFTR modulator (potentiator and corrector combination)
TargetCFTR (Cystic Fibrosis Transmembrane Conductance Regulator)
ModalitySmall molecule
Therapeutic areaPulmonary/Respiratory
PhaseFDA-approved

Mechanism of action

Ivacaftor works by binding to and stabilizing the open state of CFTR channels, allowing increased chloride ion transport across cell membranes. Lumacaftor acts as a corrector by assisting in proper folding and trafficking of CFTR protein to the cell membrane. Together, these agents restore CFTR function in cystic fibrosis patients, improving mucociliary clearance and reducing lung inflammation.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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