Last reviewed · How we verify

IV N-Acetyl Cystein

Imam Khomeini Hospital · Phase 3 active Small molecule

N-Acetyl Cysteine (NAC) replenishes intracellular glutathione and acts as a potent antioxidant to reduce oxidative stress and inflammation.

N-Acetyl Cysteine (NAC) replenishes intracellular glutathione and acts as a potent antioxidant to reduce oxidative stress and inflammation. Used for Acetaminophen (paracetamol) overdose and hepatotoxicity prevention, Acute respiratory distress syndrome (ARDS), Chronic obstructive pulmonary disease (COPD) exacerbations.

At a glance

Generic nameIV N-Acetyl Cystein
SponsorImam Khomeini Hospital
Drug classAntioxidant / Mucolytic agent
TargetGlutathione synthesis pathway; free radicals and reactive oxygen species
ModalitySmall molecule
Therapeutic areaCritical Care / Respiratory / Toxicology
PhasePhase 3

Mechanism of action

NAC is a precursor to the tripeptide glutathione, a major cellular antioxidant that neutralizes reactive oxygen species and free radicals. By restoring depleted glutathione pools, NAC protects cells from oxidative damage, reduces inflammatory cytokine production, and may enhance mucosal and tissue repair. IV administration provides rapid systemic bioavailability for acute conditions where oral absorption is insufficient.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: