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Incretin Effect

University of Campania Luigi Vanvitelli · FDA-approved active Small molecule

The incretin effect enhances insulin secretion in response to oral glucose intake by stimulating incretin hormones (GLP-1 and GIP) released from the intestine.

The incretin effect enhances insulin secretion in response to oral glucose intake by stimulating incretin hormones (GLP-1 and GIP) released from the intestine. Used for Type 2 diabetes mellitus.

At a glance

Generic nameIncretin Effect
SponsorUniversity of Campania Luigi Vanvitelli
Drug classIncretin-based therapy (mechanism class)
TargetGLP-1 receptor and/or GIP receptor
ModalitySmall molecule
Therapeutic areaDiabetes
PhaseFDA-approved

Mechanism of action

When glucose is consumed orally, intestinal L-cells release incretin hormones (glucagon-like peptide-1 [GLP-1] and glucose-dependent insulinotropic polypeptide [GIP]), which account for 50-70% of the postprandial insulin secretion. These hormones bind to their respective receptors on pancreatic beta cells, triggering glucose-dependent insulin release. This physiological mechanism is leveraged therapeutically by incretin-based drugs to improve glycemic control in type 2 diabetes.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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