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Ginger-ibuprofen

Ferrosan AS · FDA-approved active Small molecule

Ibuprofen inhibits cyclooxygenase (COX) enzymes to reduce prostaglandin synthesis and inflammation, while ginger provides additional anti-inflammatory and analgesic effects through multiple pathways.

Ibuprofen inhibits cyclooxygenase (COX) enzymes to reduce prostaglandin synthesis and inflammation, while ginger provides additional anti-inflammatory and analgesic effects through multiple pathways. Used for Pain and inflammation (general indication for marketed NSAID-ginger combination).

At a glance

Generic nameGinger-ibuprofen
SponsorFerrosan AS
Drug classNSAID with herbal adjunct
TargetCOX-1, COX-2 (ibuprofen component); multiple inflammatory pathways (ginger component)
ModalitySmall molecule
Therapeutic areaPain management, Inflammation
PhaseFDA-approved

Mechanism of action

Ibuprofen is a nonsteroidal anti-inflammatory drug (NSAID) that blocks COX-1 and COX-2 enzymes, thereby reducing prostaglandin production and decreasing inflammation, pain, and fever. Ginger contains gingerols and shogaols that inhibit inflammatory mediators and may enhance the anti-inflammatory effect. This combination product leverages both mechanisms to provide analgesic and anti-inflammatory benefits.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results