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Gilteritinib + Azacitidine + Venetoclax

Institute of Hematology & Blood Diseases Hospital, China · Phase 3 active Small molecule

This triple combination targets acute myeloid leukemia by inhibiting FLT3 signaling (gilteritinib), promoting DNA methylation-dependent differentiation (azacitidine), and blocking anti-apoptotic BCL-2 proteins (venetoclax).

This triple combination targets acute myeloid leukemia by inhibiting FLT3 signaling (gilteritinib), promoting DNA methylation-dependent differentiation (azacitidine), and blocking anti-apoptotic BCL-2 proteins (venetoclax). Used for Acute myeloid leukemia (AML), including FLT3-mutated disease.

At a glance

Generic nameGilteritinib + Azacitidine + Venetoclax
SponsorInstitute of Hematology & Blood Diseases Hospital, China
Drug classFLT3 inhibitor + hypomethylating agent + BCL-2 inhibitor combination
TargetFLT3, DNA methyltransferase, BCL-2
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Gilteritinib is a selective FLT3 inhibitor that blocks proliferative signaling in FLT3-mutated AML. Azacitidine is a hypomethylating agent that restores normal gene expression and induces differentiation. Venetoclax is a BCL-2 inhibitor that promotes apoptosis by disrupting mitochondrial survival pathways. The combination targets multiple pathways implicated in AML pathogenesis and chemoresistance.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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