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Full dose of DOAC

University Hospital, Brest · Phase 3 active Small molecule

Direct oral anticoagulants (DOACs) inhibit specific clotting factors to prevent blood clot formation.

Direct oral anticoagulants (DOACs) inhibit specific clotting factors to prevent blood clot formation. Used for Atrial fibrillation for stroke prevention, Venous thromboembolism (deep vein thrombosis and pulmonary embolism) treatment and prevention, Acute coronary syndrome (in combination with antiplatelet therapy).

At a glance

Generic nameFull dose of DOAC
SponsorUniversity Hospital, Brest
Drug classDirect oral anticoagulant (DOAC)
TargetFactor Xa or Factor IIa (thrombin)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

DOACs are a class of anticoagulants that work by directly inhibiting either Factor Xa (apixaban, rivaroxaban, edoxaban) or Factor IIa/thrombin (dabigatran), key enzymes in the coagulation cascade. By blocking these factors, DOACs prevent the formation of thrombin and subsequent fibrin clot formation, reducing thrombotic events while maintaining some residual coagulation capacity.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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