Last reviewed · How we verify

Divalproex (DV)

Weill Medical College of Cornell University · FDA-approved active Small molecule

Divalproex inhibits histone deacetylase and enhances GABA neurotransmission, leading to increased inhibitory signaling in the central nervous system.

Divalproex inhibits histone deacetylase and enhances GABA neurotransmission, leading to increased inhibitory signaling in the central nervous system. Used for Epilepsy and seizure disorders, Bipolar disorder (acute mania), Migraine prophylaxis.

At a glance

Generic nameDivalproex (DV)
Also known asValproate
SponsorWeill Medical College of Cornell University
Drug classHistone deacetylase inhibitor; anticonvulsant; mood stabilizer
TargetHistone deacetylase; GABA metabolism; sodium and calcium channels
ModalitySmall molecule
Therapeutic areaNeurology; Psychiatry
PhaseFDA-approved

Mechanism of action

Divalproex is a compound of sodium valproate and valproic acid that works through multiple mechanisms: it inhibits histone deacetylase enzymes (affecting gene expression), increases GABA synthesis and reduces GABA catabolism (enhancing inhibitory neurotransmission), and may modulate sodium and calcium channels. These combined effects produce anticonvulsant, mood-stabilizing, and neuroprotective properties.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: