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Dichloroacetate (DCA)

University of Florida · Phase 3 active Small molecule

Dichloroacetate inhibits pyruvate dehydrogenase kinase, shifting cellular metabolism from glycolysis toward oxidative phosphorylation.

Dichloroacetate inhibits pyruvate dehydrogenase kinase, shifting cellular metabolism from glycolysis toward oxidative phosphorylation. Used for Glioblastoma multiforme, Recurrent or progressive glioblastoma.

At a glance

Generic nameDichloroacetate (DCA)
Also known asgenotype, Sodium Dichloroacetate
SponsorUniversity of Florida
Drug classPyruvate dehydrogenase kinase inhibitor
TargetPyruvate dehydrogenase kinase (PDK)
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

DCA activates pyruvate dehydrogenase complex by inhibiting its inhibitory kinase, promoting the conversion of pyruvate to acetyl-CoA and increasing aerobic metabolism. This metabolic shift reduces lactate production and may enhance mitochondrial function, with potential anti-cancer effects by reducing the Warburg effect (cancer cells' reliance on glycolysis) and promoting apoptosis in tumor cells.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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