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Decitabine in combination with R-CHOP

Second Affiliated Hospital of Nanchang University · Phase 3 active Small molecule

Decitabine inhibits DNA methyltransferase to reactivate silenced tumor suppressor genes, while R-CHOP chemotherapy directly kills lymphoma cells through multiple mechanisms.

Decitabine inhibits DNA methyltransferase to reactivate silenced tumor suppressor genes, while R-CHOP chemotherapy directly kills lymphoma cells through multiple mechanisms. Used for Lymphoid malignancies (likely diffuse large B-cell lymphoma or other non-Hodgkin lymphomas).

At a glance

Generic nameDecitabine in combination with R-CHOP
SponsorSecond Affiliated Hospital of Nanchang University
Drug classHypomethylating agent combined with chemotherapy regimen
TargetDNA methyltransferase (decitabine component); CD20, topoisomerase II, microtubule dynamics (R-CHOP components)
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Decitabine is a hypomethylating agent that reverses aberrant DNA methylation in cancer cells, restoring expression of epigenetically silenced genes. R-CHOP is a standard chemotherapy regimen (rituximab, cyclophosphamide, doxorubicin, vincristine, prednisone) that combines immunotherapy with cytotoxic chemotherapy. The combination aims to enhance anti-tumor efficacy in lymphoid malignancies by synergizing epigenetic reactivation with direct cytotoxic effects.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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