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Decitabine combined with HAAG Regimen

The First Affiliated Hospital of Soochow University · Phase 3 active Small molecule

Decitabine combined with HAAG Regimen is a Hypomethylating agent combined with chemotherapy regimen Small molecule drug developed by The First Affiliated Hospital of Soochow University. It is currently in Phase 3 development for Acute myeloid leukemia (AML), particularly in elderly or relapsed/refractory patients, Myelodysplastic syndrome (MDS).

Decitabine inhibits DNA methyltransferase to reactivate silenced tumor suppressor genes, combined with HAAG chemotherapy (cytarabine, aclarubicin, amsacrine, granulocyte colony-stimulating factor) to enhance cytotoxic effects in hematologic malignancies.

Decitabine inhibits DNA methyltransferase to reactivate silenced tumor suppressor genes, combined with HAAG chemotherapy (cytarabine, aclarubicin, amsacrine, granulocyte colony-stimulating factor) to enhance cytotoxic effects in hematologic malignancies. Used for Acute myeloid leukemia (AML), particularly in elderly or relapsed/refractory patients, Myelodysplastic syndrome (MDS).

At a glance

Generic nameDecitabine combined with HAAG Regimen
SponsorThe First Affiliated Hospital of Soochow University
Drug classHypomethylating agent combined with chemotherapy regimen
TargetDNA methyltransferase (DNMT); also targets topoisomerase II and DNA synthesis
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Decitabine is a hypomethylating agent that removes aberrant DNA methylation marks on tumor suppressor genes, restoring their expression and inducing differentiation and apoptosis in leukemic cells. The HAAG regimen provides intensive chemotherapy with multiple mechanisms of action. Together, this combination targets both epigenetic dysregulation and provides direct cytotoxic activity against leukemic blasts.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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Frequently asked questions about Decitabine combined with HAAG Regimen

What is Decitabine combined with HAAG Regimen?

Decitabine combined with HAAG Regimen is a Hypomethylating agent combined with chemotherapy regimen drug developed by The First Affiliated Hospital of Soochow University, indicated for Acute myeloid leukemia (AML), particularly in elderly or relapsed/refractory patients, Myelodysplastic syndrome (MDS).

How does Decitabine combined with HAAG Regimen work?

Decitabine inhibits DNA methyltransferase to reactivate silenced tumor suppressor genes, combined with HAAG chemotherapy (cytarabine, aclarubicin, amsacrine, granulocyte colony-stimulating factor) to enhance cytotoxic effects in hematologic malignancies.

What is Decitabine combined with HAAG Regimen used for?

Decitabine combined with HAAG Regimen is indicated for Acute myeloid leukemia (AML), particularly in elderly or relapsed/refractory patients, Myelodysplastic syndrome (MDS).

Who makes Decitabine combined with HAAG Regimen?

Decitabine combined with HAAG Regimen is developed by The First Affiliated Hospital of Soochow University (see full The First Affiliated Hospital of Soochow University pipeline at /company/the-first-affiliated-hospital-of-soochow-university).

What drug class is Decitabine combined with HAAG Regimen in?

Decitabine combined with HAAG Regimen belongs to the Hypomethylating agent combined with chemotherapy regimen class. See all Hypomethylating agent combined with chemotherapy regimen drugs at /class/hypomethylating-agent-combined-with-chemotherapy-regimen.

What development phase is Decitabine combined with HAAG Regimen in?

Decitabine combined with HAAG Regimen is in Phase 3.

What are the side effects of Decitabine combined with HAAG Regimen?

Common side effects of Decitabine combined with HAAG Regimen include Myelosuppression (neutropenia, thrombocytopenia, anemia), Infection, Nausea and vomiting, Mucositis, Hepatotoxicity, Tumor lysis syndrome.

What does Decitabine combined with HAAG Regimen target?

Decitabine combined with HAAG Regimen targets DNA methyltransferase (DNMT); also targets topoisomerase II and DNA synthesis and is a Hypomethylating agent combined with chemotherapy regimen.

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