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Cyclosporins/Tacrolimus

University of Minnesota · FDA-approved active Small molecule

Cyclosporins and tacrolimus suppress the immune system by inhibiting calcineurin, a key enzyme required for T-cell activation and cytokine production.

Cyclosporins and tacrolimus suppress the immune system by inhibiting calcineurin, a key enzyme required for T-cell activation and cytokine production. Used for Prevention of organ rejection in transplant recipients (kidney, heart, liver, pancreas), Autoimmune diseases including atopic dermatitis, psoriasis, and rheumatoid arthritis, Graft-versus-host disease (GVHD) prophylaxis and treatment.

At a glance

Generic nameCyclosporins/Tacrolimus
Also known asGengraf (cyclosporin), Neoral (cyclosporin), Prograf (tacrolimus)
SponsorUniversity of Minnesota
Drug classCalcineurin inhibitor
TargetCalcineurin
ModalitySmall molecule
Therapeutic areaImmunology
PhaseFDA-approved

Mechanism of action

Both drugs bind to immunophilins (cyclophilin for cyclosporine, FKBP for tacrolimus) and form complexes that inhibit calcineurin phosphatase activity. This prevents dephosphorylation and nuclear translocation of NFAT (nuclear factor of activated T cells), blocking the transcription of IL-2 and other pro-inflammatory cytokines essential for T-cell proliferation and immune response.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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