Last reviewed · How we verify

CSL312

CSL Behring · Phase 3 active Biologic

CSL312 is an apolipoprotein A-I (apoA-I) mimetic that increases HDL cholesterol and promotes reverse cholesterol transport.

CSL312 is an apolipoprotein A-I (apoA-I) mimetic that increases HDL cholesterol and promotes reverse cholesterol transport. Used for Acute coronary syndrome with elevated lipoprotein(a), Cardiovascular risk reduction in patients with low HDL cholesterol.

At a glance

Generic nameCSL312
Also known asGaradacimab, Factor XIIa inhibitor monoclonal antibody, garadacimab, Factor XIIa antagonist monoclonal antibody, Gardacimab
SponsorCSL Behring
Drug classapoA-I mimetic
TargetApolipoprotein A-I pathway; ABCA1 transporter
ModalityBiologic
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

CSL312 is designed to mimic the structure and function of apolipoprotein A-I, the major protein component of HDL particles. By enhancing HDL-mediated reverse cholesterol transport, the drug aims to reduce atherosclerotic cardiovascular disease risk. It works by facilitating the removal of cholesterol from peripheral tissues and atherosclerotic plaques, promoting their transport to the liver for excretion.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: