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Cocaine N-acetylcysteine

Hospital de Clinicas de Porto Alegre · FDA-approved active Small molecule

N-acetylcysteine replenishes glutathione stores and provides antioxidant protection to counteract cocaine-induced oxidative stress and toxicity.

N-acetylcysteine replenishes glutathione stores and provides antioxidant protection to counteract cocaine-induced oxidative stress and toxicity. Used for Cocaine use disorder and cocaine toxicity mitigation.

At a glance

Generic nameCocaine N-acetylcysteine
Also known asNAC
SponsorHospital de Clinicas de Porto Alegre
Drug classAntioxidant / Glutathione precursor
TargetGlutathione synthesis pathway; indirect antioxidant effects
ModalitySmall molecule
Therapeutic areaAddiction Medicine / Toxicology
PhaseFDA-approved

Mechanism of action

Cocaine causes acute oxidative stress and depletion of the antioxidant glutathione in tissues, particularly in the brain and cardiovascular system. N-acetylcysteine (NAC) is a glutathione precursor that restores cellular antioxidant capacity and reduces reactive oxygen species, thereby mitigating cocaine-related cellular damage and potentially reducing addiction-related neurobiological changes.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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