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cisplatin concurrent chemotherapy

Sun Yat-sen University · Phase 3 active Small molecule Quality 60/100

cisplatin concurrent chemotherapy is a Platinum-based chemotherapy agent Small molecule drug developed by Sun Yat-sen University. It is currently in Phase 3 development.

Cisplatin forms DNA crosslinks that inhibit DNA replication and transcription, leading to cancer cell death, while also acting as a radiosensitizer.

Likelihood of approval
58.3% vs 58.3% industry baseline
If approved by FDA: likely 2028–2030
Steps remaining: NDA/BLA submission
Confidence: High
Why this estimate
  • Baseline phase 3 → approval rate +58.3pp
    Industry-wide phase 3 drugs reach approval ~58.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2028–2030
EMA EU 2029–2031 +0.7 yr
MHRA GB 2029–2031 +0.7 yr
Health Canada CA 2029–2032 +0.9 yr
TGA AU 2029–2032 +1.2 yr
PMDA JP 2029–2032 +1.5 yr
NMPA CN 2030–2033 +2.3 yr
MFDS KR 2029–2032 +1.4 yr
CDSCO IN 2029–2033 +1.8 yr
ANVISA BR 2030–2033 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic namecisplatin concurrent chemotherapy
SponsorSun Yat-sen University
Drug classPlatinum-based chemotherapy agent
ModalitySmall molecule
PhasePhase 3

Mechanism of action

Cisplatin binds to DNA forming intrastrand and interstrand crosslinks, primarily at guanine bases. This prevents DNA unwinding and replication, triggering apoptosis in rapidly dividing cancer cells. When given concurrently with radiation, cisplatin enhances radiation-induced DNA damage and impairs DNA repair mechanisms.

Approved indications

Common side effects

No common side effects on file.

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about cisplatin concurrent chemotherapy

What is cisplatin concurrent chemotherapy?

cisplatin concurrent chemotherapy is a Platinum-based chemotherapy agent drug developed by Sun Yat-sen University.

How does cisplatin concurrent chemotherapy work?

Cisplatin forms DNA crosslinks that inhibit DNA replication and transcription, leading to cancer cell death, while also acting as a radiosensitizer.

Who makes cisplatin concurrent chemotherapy?

cisplatin concurrent chemotherapy is developed by Sun Yat-sen University (see full Sun Yat-sen University pipeline at /company/sun-yat-sen-university).

What drug class is cisplatin concurrent chemotherapy in?

cisplatin concurrent chemotherapy belongs to the Platinum-based chemotherapy agent class. See all Platinum-based chemotherapy agent drugs at /class/platinum-based-chemotherapy-agent.

What development phase is cisplatin concurrent chemotherapy in?

cisplatin concurrent chemotherapy is in Phase 3.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing