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cisplatin + capecitabine

AstraZeneca · Phase 3 active Small molecule

Cisplatin crosslinks DNA to prevent replication while capecitabine is converted to fluorouracil to inhibit thymidylate synthase, together blocking cancer cell division and survival.

Cisplatin crosslinks DNA to prevent replication while capecitabine is converted to fluorouracil to inhibit thymidylate synthase, together blocking cancer cell division and survival. Used for Metastatic gastric cancer, Metastatic esophageal cancer, Advanced biliary tract cancer.

At a glance

Generic namecisplatin + capecitabine
SponsorAstraZeneca
Drug classChemotherapy combination (platinum agent + antimetabolite)
TargetDNA (cisplatin); thymidylate synthase (capecitabine)
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Cisplatin is a platinum-based alkylating agent that forms covalent DNA adducts, causing interstrand and intrastrand crosslinks that prevent DNA replication and transcription. Capecitabine is a prodrug of fluorouracil that inhibits thymidylate synthase, disrupting nucleotide synthesis and DNA replication. The combination provides synergistic cytotoxic effects against rapidly dividing cancer cells.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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