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Chemodenervation (4 months)

University of Wisconsin, Madison · FDA-approved active Small molecule

Chemodenervation uses botulinum toxin to temporarily paralyze muscles by blocking acetylcholine release at the neuromuscular junction.

Chemodenervation uses botulinum toxin to temporarily paralyze muscles by blocking acetylcholine release at the neuromuscular junction. Used for Muscle spasticity and hypertonicity, Dystonia, Migraine prophylaxis.

At a glance

Generic nameChemodenervation (4 months)
Also known asonabotulinumtoxin A, Botox, incobotulinumtoxin A, Xeomin
SponsorUniversity of Wisconsin, Madison
Drug classBotulinum toxin
TargetSNARE complex (synaptobrevin, SNAP-25, syntaxin)
ModalitySmall molecule
Therapeutic areaNeurology, Dermatology, Rehabilitation Medicine
PhaseFDA-approved

Mechanism of action

Botulinum toxin cleaves SNARE proteins required for acetylcholine vesicle release, preventing muscle contraction. The effect is temporary, lasting approximately 3–4 months, after which neuromuscular function gradually recovers as new nerve terminals form. This mechanism is exploited therapeutically to reduce muscle hyperactivity in various conditions.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results