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Canesten®

Ache Laboratorios Farmaceuticos S.A. · FDA-approved active Small molecule

Canesten inhibits fungal cell membrane synthesis by blocking ergosterol production through inhibition of lanosterol 14α-demethylase.

Canesten inhibits fungal cell membrane synthesis by blocking ergosterol production through inhibition of lanosterol 14α-demethylase. Used for Vaginal candidiasis, Dermatophyte infections (tinea pedis, tinea corporis, tinea cruris), Cutaneous candidiasis.

At a glance

Generic nameCanesten®
Also known asclotrimazole
SponsorAche Laboratorios Farmaceuticos S.A.
Drug classImidazole antifungal
TargetLanosterol 14α-demethylase (CYP51)
ModalitySmall molecule
Therapeutic areaInfectious Disease
PhaseFDA-approved

Mechanism of action

Canesten contains clotrimazole, an imidazole antifungal that disrupts the synthesis of ergosterol, a critical component of fungal cell membranes. By inhibiting the cytochrome P450-dependent enzyme lanosterol 14α-demethylase, clotrimazole causes accumulation of toxic sterol precursors and membrane instability, leading to fungal cell death. This mechanism is selective for fungal cells over human cells due to differences in sterol composition.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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