Last reviewed · How we verify

Botulinum Toxin A - Open Label

Croma-Pharma GmbH · Phase 3 active Small molecule

Botulinum Toxin A blocks acetylcholine release at the neuromuscular junction by cleaving SNARE proteins, causing temporary muscle paralysis.

Botulinum Toxin A blocks acetylcholine release at the neuromuscular junction by cleaving SNARE proteins, causing temporary muscle paralysis. Used for Cervical dystonia, Blepharospasm, Hemifacial spasm.

At a glance

Generic nameBotulinum Toxin A - Open Label
Also known asBoNT/A-DP
SponsorCroma-Pharma GmbH
Drug classNeurotoxin; Acetylcholine release inhibitor
TargetSNAP-25 (synaptosome-associated protein of 25 kDa)
ModalitySmall molecule
Therapeutic areaNeurology; Aesthetics; Pain Management
PhasePhase 3

Mechanism of action

Botulinum Toxin A is a neurotoxin that irreversibly cleaves SNARE proteins (specifically SNAP-25) required for acetylcholine vesicle fusion and release at the presynaptic terminal. This prevents neuromuscular transmission, resulting in temporary flaccid paralysis of injected muscles. The effect is reversible over 3-4 months as new nerve terminals form and acetylcholine signaling is restored.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: