Last reviewed · How we verify

BOTOX ® 200 Unités

University Hospital, Clermont-Ferrand · Phase 3 active Small molecule

BOTOX (botulinum toxin type A) blocks acetylcholine release at the neuromuscular junction, causing temporary muscle paralysis.

BOTOX (botulinum toxin type A) blocks acetylcholine release at the neuromuscular junction, causing temporary muscle paralysis. Used for Cervical dystonia, Blepharospasm, Hemifacial spasm.

At a glance

Generic nameBOTOX ® 200 Unités
SponsorUniversity Hospital, Clermont-Ferrand
Drug classNeurotoxin; botulinum toxin type A
TargetSNARE complex (specifically cleaves SNAP-25)
ModalitySmall molecule
Therapeutic areaNeurology; Aesthetics; Pain Management
PhasePhase 3

Mechanism of action

Botulinum toxin type A cleaves SNARE proteins required for acetylcholine vesicle fusion and release at the presynaptic terminal. This results in flaccid paralysis of the injected muscle that typically lasts 3-4 months. The effect is reversible as the body gradually regenerates the cleaved proteins and forms new neuromuscular junctions.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results