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Aspirin loading dose

Icahn School of Medicine at Mount Sinai · FDA-approved active Small molecule

Aspirin irreversibly inhibits cyclooxygenase (COX) enzymes to reduce thromboxane A2 production and platelet aggregation.

Aspirin irreversibly inhibits cyclooxygenase (COX) enzymes to reduce thromboxane A2 production and platelet aggregation. Used for Acute coronary syndrome (ACS) and myocardial infarction, Stroke prevention in patients with prior ischemic stroke or transient ischemic attack, Stable coronary artery disease.

At a glance

Generic nameAspirin loading dose
Also known asAcetylsalicylic acid, ASA
SponsorIcahn School of Medicine at Mount Sinai
Drug classAntiplatelet agent; nonsteroidal anti-inflammatory drug (NSAID)
TargetCyclooxygenase-1 (COX-1); Cyclooxygenase-2 (COX-2)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

Aspirin acetylates serine residues on COX-1 and COX-2 enzymes, permanently blocking their catalytic activity. This prevents the synthesis of thromboxane A2 in platelets, a potent platelet activator and vasoconstrictor, thereby reducing platelet aggregation and thrombotic events. A loading dose delivers a rapid, high concentration to achieve quick antiplatelet effects, particularly important in acute coronary syndromes.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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