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ASA evening

Basque Health Service · Phase 3 active Small molecule

Aspirin inhibits platelet aggregation by irreversibly acetylating cyclooxygenase (COX) enzymes, reducing thromboxane A2 production.

Aspirin inhibits platelet aggregation by irreversibly acetylating cyclooxygenase (COX) enzymes, reducing thromboxane A2 production. Used for Secondary prevention of cardiovascular events (myocardial infarction, stroke, transient ischemic attack), Primary prevention of cardiovascular disease in high-risk patients.

At a glance

Generic nameASA evening
Also known asASA, Low dose, aspirine
SponsorBasque Health Service
Drug classNonsteroidal anti-inflammatory drug (NSAID); antiplatelet agent
TargetCyclooxygenase-1 (COX-1), Cyclooxygenase-2 (COX-2)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

Aspirin (acetylsalicylic acid) covalently acetylates COX-1 and COX-2 enzymes in platelets, preventing the synthesis of thromboxane A2, a potent platelet activator and vasoconstrictor. This antiplatelet effect reduces clot formation and is the basis for its use in cardiovascular disease prevention. Evening dosing may optimize circadian effects on platelet function and thrombotic risk.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results