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Arsenic Trioxide Injectable Solution

Medsenic · Phase 2 active Small molecule

Arsenic trioxide induces differentiation and apoptosis in leukemic cells, particularly in acute promyelocytic leukemia (APL), through multiple pathways including PML-RARA protein degradation and reactive oxygen species generation.

Arsenic trioxide induces differentiation and apoptosis in leukemic cells, particularly in acute promyelocytic leukemia (APL), through multiple pathways including PML-RARA protein degradation and reactive oxygen species generation. Used for Acute promyelocytic leukemia (APL), Relapsed or refractory APL.

At a glance

Generic nameArsenic Trioxide Injectable Solution
Also known asTrisenox / Arscimed
SponsorMedsenic
Drug classArsenic compound; differentiation agent and apoptosis inducer
TargetPML-RARA fusion protein; mitochondrial apoptotic pathway
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 2

Mechanism of action

Arsenic trioxide targets the PML-RARA fusion protein characteristic of APL, causing its degradation and leading to differentiation of leukemic promyelocytes into mature granulocytes. It also triggers apoptosis through mitochondrial pathways and generation of reactive oxygen species. The drug has dual activity as both a differentiating agent and an apoptosis inducer, making it effective in APL and potentially other hematologic malignancies.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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