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Aprepitant+palonosetron+dexamethasone

Union Hospital, Tongji Medical College, Huazhong University of Science and Technology · Phase 3 active Small molecule

This combination blocks chemotherapy-induced nausea and vomiting (CINV) through three complementary pathways: NK1 receptor antagonism, 5-HT3 receptor antagonism, and glucocorticoid anti-inflammatory effects.

This combination blocks chemotherapy-induced nausea and vomiting (CINV) through three complementary pathways: NK1 receptor antagonism, 5-HT3 receptor antagonism, and glucocorticoid anti-inflammatory effects. Used for Chemotherapy-induced nausea and vomiting (CINV) prevention in patients receiving highly emetogenic chemotherapy.

At a glance

Generic nameAprepitant+palonosetron+dexamethasone
Also known asPDA
SponsorUnion Hospital, Tongji Medical College, Huazhong University of Science and Technology
Drug classAntiemetic combination (NK1 antagonist + 5-HT3 antagonist + corticosteroid)
TargetNK1 receptor, 5-HT3 receptor, glucocorticoid receptor
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Aprepitant is a neurokinin-1 (NK1) receptor antagonist that blocks substance P signaling in the chemoreceptor trigger zone and vomiting center. Palonosetron is a 5-HT3 receptor antagonist that blocks serotonin signaling in the chemoreceptor trigger zone. Dexamethasone is a corticosteroid that provides additional antiemetic effects through multiple mechanisms including glucocorticoid receptor activation. Together, these agents provide comprehensive CINV prophylaxis across acute, delayed, and breakthrough phases.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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